About this trial
Nonspecific low back pain affects approximately 80% of the population at some point in their lives.
The psoas muscle-positioned between the lumbar and pelvic regions-together with the paravertebral muscles, constitutes the primary muscular group responsible for dynamic stabilization of the spine. Alterations in this muscle may contribute to the onset of low back pain by modifying spinal biomechanics, particularly through increased lumbar lordosis and greater mechanical overload in the lumbar region.
Latent myofascial trigger points are asymptomatic but can lead to muscle shortening and weakness. They are characterized by local tenderness and may provoke a localized muscular response known as a local twitch response.
The aim of this randomized experimental study was to evaluate the immediate effect of ischemic compression applied to the trigger point of the psoas muscle on pain perception and lumbar range of motion.
Researchers will compare a group with a placebo technique to see if the release of the psoas muscle produces effects in this condition.
The variables analyzed in this study included low back pain, assessed using the Numeric Pain Rating Scale (NPRS); lateral lumbar flexion (right and left), measured using a tape measure (distance from the third finger to the floor); and lumbar flexion range of motion, assessed using the Schober test.
Eligibility criteria
Qualifiers
Age between 18 and 30 years.
Presence of low back pain for less than 1 month.
Presence of a latent trigger point in the psoas muscle.
Disqualifiers
Volunteers with low back pain caused by: Disc herniation, Tumor, Infection, Fracture, Osteoporosis, Structural deformity or Inflammation.
Individuals with a history of spinal surgery.
Continuous use of analgesics.
Participants who had undergone manual therapy in the previous month.
Trial design
Treatments tested in this trial
- Trigger point release
- Simulation of the intestinal listening technique.